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PubMed ID:
18279672
Public Release Type:
Journal
Publication Year: 2008
Affiliation: Department of Internal Medicine, University of Texas Southwestern Medical Center, 5959 Harry Hines Boulevard, Dallas, TX 75390, USA.
DOI:
https://doi.org/10.1016/j.cca.2008.01.018
Authors:
Lalani E,
Smith A,
Barakat F,
Hassenein T,
Campbell M,
Reddy R,
Taylor W,
Satyanarayana R,
Prosser C,
Rossaro L,
Salvatori J,
Stravitz T,
Misra C,
Munoz S,
Huntley N,
Reuben A,
Rush R,
Harrison ME,
Senkbeil L,
Schilsky M,
Brown R Jr,
Casson D,
Chung R,
Avant L,
McGuire B,
Welch S,
Fontana RJ,
Peacock V,
Han S,
Ingram K,
Schwartz J,
Zaman A,
Gottstein J,
Blei AT,
Morton D,
Shakil AO,
Coultrup S,
Murray N,
Groettum C,
Hay JE,
Bernard T,
McCashland TM,
Emre S,
Partovi K,
Davern TJ,
Gerstle L,
Crippin JS,
Do H,
Larson AM,
Reisch JS,
Hynan LS,
Pezzia C,
Polson J,
Lee WM,
Acute Liver Failure Study Group,
Lee WM,
Hynan LS,
Balko JA,
Khan AI,
Koff JM,
Murray NG,
Fuller D,
Orsulak P,
Wians FH Jr,
Polson J
Studies:
Acute Liver Failure Study Group: Adult Acute Liver Failure Study
Acetaminophen toxicity is the most common form of acute liver failure in the U.S. After acetaminophen overdoses, quantitation of plasma acetaminophen can aid in predicting severity of injury. However, recent case reports have suggested that acetaminophen concentrations may be falsely increased in the presence of hyperbilirubinemia.
