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Publication Information
Affiliation
Department of Pathology, Cleveland Clinic, Cleveland, OH, USA.; Department of Medicine, Indiana University School of Medicine, Indianapolis, IN, USA.; Department of Pathology, Indiana University School of Medicine, Indianapolis, IN, USA.; Center for Clinical Trials, The Johns Hopkins University, Bloomberg School of Public Health, Baltimore, MD, USA.; Center for Clinical Trials, The Johns Hopkins University, Bloomberg School of Public Health, Baltimore, MD, USA.; Center for Clinical Trials, The Johns Hopkins University, Bloomberg School of Public Health, Baltimore, MD, USA.; Department of Pathology, Pacific Rim Pathology, San Diego, CA, USA.; Department of Pathology, Saint Louis University, St Louis, MI, USA.; Department of Pathology, University of California San Francisco, San Francisco, CA, USA.; Laboratory of Pathology, National Cancer Institute, Bethesda, MD, USA.; Department of Pathology, University of Washington School of Medicine, Seattle, WA, USA.; Department of Medicine, Indiana University School of Medicine, Indianapolis, IN, USA.; Department of Pathology, Duke University, Durham, NC, USA.
Authors
Allende Daniela S, Gawrieh Samer, Cummings Oscar W, Belt Patricia, Wilson Laura, Van Natta Mark, Behling Cynthia A, Carpenter Danielle, Gill Ryan M, Kleiner David E, Yeh Mathew M, Chalasani Naga, Guy Cynthia D
Studies
Abstract
Glycogen synthesis and storage are normal hepatocyte functions. However, glycogenosis, defined as excess hepatocyte glycogen visible by routine H&E light microscopy, has not been well characterized in nonalcoholic fatty liver disease (NAFLD).